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Weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in Saccharomyces cerevisiae

Weak organic acids are used as food preservatives to inhibit the growth of spoilage yeasts, including Saccharomyces cerevisiae. Long-term adaptation to weak acids requires the increased expression of the ATP-binding cassette transporter Pdr12p, which catalyses the active efflux of the weak acids fro...

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Päätekijät: Mollapour, Mehdi, Phelan, John P., Millson, Stefan H., Piper, Peter W., Cooke, Frank T.
Aineistotyyppi: Artikkeli
Kieli:English
Julkaistu: Portland Press Ltd. 2006
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Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC1409697/
https://ncbi.nlm.nih.gov/pubmed/16316315
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/BJ20051765
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spelling pubmed-14096972006-10-01 Weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in Saccharomyces cerevisiae Mollapour, Mehdi Phelan, John P. Millson, Stefan H. Piper, Peter W. Cooke, Frank T. Biochem J Research Article Weak organic acids are used as food preservatives to inhibit the growth of spoilage yeasts, including Saccharomyces cerevisiae. Long-term adaptation to weak acids requires the increased expression of the ATP-binding cassette transporter Pdr12p, which catalyses the active efflux of the weak acids from the cytosol; however, very little is known about the signalling events immediately following application of weak acid stress. We have investigated the effects of weak acids on two stress-responsive signalling molecules, PtdIns(3,5)P(2) and PtdIns(4,5)P(2), which in S. cerevisiae are synthesized by Fab1p and Mss4p respectively. At low extracellular pH, benzoic acid, sorbic acid and acetic acid all cause a transient reduction in PtdIns(3,5)P(2) accumulation and a more persistent rise in PtdIns(4,5)P(2) levels. The increase in PtdIns(4,5)P(2) levels is accompanied by a reorganization of the actin cytoskeleton. However, changes in PtdInsP(2) levels are independent of weak acid-induced Pdr12p expression. In contrast, changing the extracellular medium to alkaline pH provokes a prolonged and substantial rise in PtdIns(3,5)P(2) levels. As PtdIns(3,5)P(2) synthesis is required for correct vacuole acidification, it is possible that levels of this molecule are modulated to maintain intracellular pH homoeostasis in response to weak acid and alkali stresses. In conclusion, we have expanded the repertoire of stress responses that affect PtdInsP(2) levels to include weak acid and alkali stresses. Portland Press Ltd. 2006-03-15 2006-04-01 /pmc/articles/PMC1409697/ /pubmed/16316315 http://dx.doi.org/10.1042/BJ20051765 Text en The Biochemical Society, London
institution US National Library of Medicine
collection PubMed Central
language English
format Article
topic Research Article
spellingShingle Research Article
Mollapour, Mehdi
Phelan, John P.
Millson, Stefan H.
Piper, Peter W.
Cooke, Frank T.
Weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in Saccharomyces cerevisiae
description Weak organic acids are used as food preservatives to inhibit the growth of spoilage yeasts, including Saccharomyces cerevisiae. Long-term adaptation to weak acids requires the increased expression of the ATP-binding cassette transporter Pdr12p, which catalyses the active efflux of the weak acids from the cytosol; however, very little is known about the signalling events immediately following application of weak acid stress. We have investigated the effects of weak acids on two stress-responsive signalling molecules, PtdIns(3,5)P(2) and PtdIns(4,5)P(2), which in S. cerevisiae are synthesized by Fab1p and Mss4p respectively. At low extracellular pH, benzoic acid, sorbic acid and acetic acid all cause a transient reduction in PtdIns(3,5)P(2) accumulation and a more persistent rise in PtdIns(4,5)P(2) levels. The increase in PtdIns(4,5)P(2) levels is accompanied by a reorganization of the actin cytoskeleton. However, changes in PtdInsP(2) levels are independent of weak acid-induced Pdr12p expression. In contrast, changing the extracellular medium to alkaline pH provokes a prolonged and substantial rise in PtdIns(3,5)P(2) levels. As PtdIns(3,5)P(2) synthesis is required for correct vacuole acidification, it is possible that levels of this molecule are modulated to maintain intracellular pH homoeostasis in response to weak acid and alkali stresses. In conclusion, we have expanded the repertoire of stress responses that affect PtdInsP(2) levels to include weak acid and alkali stresses.
author Mollapour, Mehdi
Phelan, John P.
Millson, Stefan H.
Piper, Peter W.
Cooke, Frank T.
author_facet Mollapour, Mehdi
Phelan, John P.
Millson, Stefan H.
Piper, Peter W.
Cooke, Frank T.
author_sort Mollapour, Mehdi
title Weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in Saccharomyces cerevisiae
title_short Weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in Saccharomyces cerevisiae
title_full Weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in Saccharomyces cerevisiae
title_fullStr Weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in Saccharomyces cerevisiae
title_full_unstemmed Weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in Saccharomyces cerevisiae
title_sort weak acid and alkali stress regulate phosphatidylinositol bisphosphate synthesis in saccharomyces cerevisiae
publisher Portland Press Ltd.
publisher_facet Portland Press Ltd.
publishDate 2006
url https://ncbi.nlm.nih.gov/pmc/articles/PMC1409697/
https://ncbi.nlm.nih.gov/pubmed/16316315
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/BJ20051765
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