Nitric oxide protects cultured rheumatoid synovial cells from Fas-induced apoptosis by inhibiting caspase-3

Nitric oxide (NO) is elevated in the synovial fluids and sera of patients with rheumatoid arthritis (RA) and is thought to be an important proinflammatory mediator in the rheumatoid synovium. To test the hypothesis that NO might modulate the apoptosis-inducing signal pathway, we investigated the eff...

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Bibliografske podrobnosti
Main Authors: Migita, Kiyoshi, Yamasaki, Satoshi, Kita, Masako, Ida, Hiroaki, Shibatomi, Kazutaka, Kawakami, Atsushi, Aoyagi, Takahiko, Eguchi, Katsuni
Format: Artigo
Jezik:English
Izdano: Blackwell Science Inc 2001
Teme:
Original Articles
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC1783248/
https://ncbi.nlm.nih.gov/pubmed/11454065
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1046/j.1365-2567.2001.01252.x
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Internet

https://ncbi.nlm.nih.gov/pmc/articles/PMC1783248/
https://ncbi.nlm.nih.gov/pubmed/11454065
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1046/j.1365-2567.2001.01252.x

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