Vaccinia Virus K1L and C7L Inhibit Antiviral Activities Induced by Type I Interferons

Cellular tropism of vaccinia virus (VACV) is regulated by host range genes, including K1L, C7L, and E3L. While E3L is known to support viral replication by antagonizing interferon (IFN) effectors, including PKR, the exact functions of K1L and C7L are unclear. Here, we show that K1L and C7L can also...

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Bibliografiset tiedot
Päätekijät: Meng, Xiangzhi, Jiang, Canhua, Arsenio, Janilyn, Dick, Kevin, Cao, Jingxin, Xiang, Yan
Aineistotyyppi: Artikkeli
Kieli:English
Julkaistu: American Society for Microbiology (ASM) 2009
Aiheet:
Virus-Cell Interactions
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2753149/
https://ncbi.nlm.nih.gov/pubmed/19656868
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.01260-09
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Internet

https://ncbi.nlm.nih.gov/pmc/articles/PMC2753149/
https://ncbi.nlm.nih.gov/pubmed/19656868
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.01260-09

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