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Syntaxin 1A inhibits CFTR chloride channels by means of domain-specific protein–protein interactions
Previously we showed that the functional activity of the epithelial chloride channel that is encoded by the cystic fibrosis gene (CFTR) is reciprocally modulated by two components of the vesicle fusion machinery, syntaxin 1A and Munc-18. Here we report that syntaxin 1A inhibits CFTR chloride channel...
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| Autors principals: | , , , , |
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| Format: | Article |
| Idioma: | en |
| Publicat: |
The National Academy of Sciences
1998
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC28005/ https://ncbi.nlm.nih.gov/pubmed/9724814 |
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