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Neuronal JNK pathway activation by IL-1 is mediated through IL1RAPL1, a protein required for development of cognitive functions
Interleukin-1-Receptor Accessory Protein Like 1 (IL1RAPL1) gene mutations are associated to cognitive impairment ranging from non-syndromic X-linked mental retardation to autism. Functionally IL1RAPL1 belongs to a novel family of Toll/IL-1 Receptors, but its ligand is unknown. In a recent study, we...
Tallennettuna:
| Aineistotyyppi: | Artikkeli |
|---|---|
| Kieli: | en |
| Julkaistu: |
Landes Bioscience
2010
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2918768/ https://ncbi.nlm.nih.gov/pubmed/20714405 |
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