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Cellular senescence in cancer treatment: friend or foe?
Damage to DNA, the prime target of anticancer therapy, triggers programmed cellular responses. In addition to apoptosis, therapy-mediated premature senescence has been identified as another drug-responsive program that impacts the outcome of cancer therapy. Here, we discuss whether induction of sene...
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| Formaat: | Artikel |
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American Society for Clinical Investigation
2004
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC311442/ https://ncbi.nlm.nih.gov/pubmed/14722606 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI200420784 |
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