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Transcytosis of cholera toxin subunits across model human intestinal epithelia.

Cholera toxin (CT) elicits a massive secretory response from intestinal epithelia by binding apical receptors (ganglioside GM1) and ultimately activating basolateral effectors (adenylate cyclase). The mechanism of signal transduction from apical to basolateral membrane, however, remains undefined. W...

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Detalles Bibliográficos
Main Authors: Lencer, W I, Moe, S, Rufo, P A, Madara, J L
Formato: Artigo
Idioma:en
Publicado: 1995
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC40742/
https://ncbi.nlm.nih.gov/pubmed/7479732
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