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Yeast histone H3 and H4 N termini function through different GAL1 regulatory elements to repress and activate transcription.
Previous work has shown that N-terminal deletions of yeast histone H3 cause a 2- to 4-fold increase in the induction of GAL1 and a number of other genes involved in galactose metabolism. In contrast, deletions at the H4 N terminus cause a 10- to 20-fold decrease in the induction of these same GAL ge...
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| Asıl Yazarlar: | , , |
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| Materyal Türü: | Makale |
| Dil: | en |
| Baskı/Yayın Bilgisi: |
1995
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC41757/ https://ncbi.nlm.nih.gov/pubmed/7777566 |
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