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Resistance to aspirin is increased by ST-elevation myocardial infarction and correlates with adenosine diphosphate levels

BACKGROUND: To be fully activated platelets are dependent on two positive feedback loops; the formation of thromboxane A(2 )by cyclooxygenase in the platelets and the release of ADP. We wanted to evaluate the effect of aspirin on platelet function in patients with acute coronary syndromes and we hyp...

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Detalhes bibliográficos
Main Authors: Borna, Catharina, Lazarowski, Eduardo, van Heusden, Catharina, Öhlin, Hans, Erlinge, David
Formato: Artigo
Idioma:English
Publicado em: BioMed Central 2005
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1192820/
https://ncbi.nlm.nih.gov/pubmed/16045804
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1477-9560-3-10
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