Alternative Splicing Controls the Mechanisms of FAK Autophosphorylation

Focal adhesion kinase (FAK) is activated following integrin engagement or stimulation of transmembrane receptors. Autophosphorylation of FAK on Tyr-397 is a critical event, allowing binding of Src family kinases and activation of signal transduction pathways. Tissue-specific alternative splicing gen...

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Detaylı Bibliyografya
Asıl Yazarlar: Toutant, Madeleine, Costa, Alicia, Studler, Jeanne-Marie, Kadaré, Gress, Carnaud, Michèle, Girault, Jean-Antoine
Materyal Türü: Makale
Dil:en
Baskı/Yayın Bilgisi: American Society for Microbiology 2002
Konular:
Cell Growth and Development
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC134714/
https://ncbi.nlm.nih.gov/pubmed/12391143
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.22.22.7731-7743.2002
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Internet

https://ncbi.nlm.nih.gov/pmc/articles/PMC134714/
https://ncbi.nlm.nih.gov/pubmed/12391143
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.22.22.7731-7743.2002

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